| Literature DB >> 10709668 |
P A Wood1, C L Kelly-Kurtz, M E Hinsdale, D A Hamm, W J Rhead.
Abstract
The SCAD deficient mouse model has been useful to investigate mechanisms of deficient fatty acid oxidation disease in human patients. This mouse model has been thoroughly characterized and is readily available from the Jackson Laboratory. Using the new technologies of gene-knockout mouse modeling, we envisage developing additional members of the acyl-CoA dehydrogenase family of enzyme deficiencies in mice and furthering our understanding of fatty acid metabolism in health and disease.Entities:
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Year: 1999 PMID： 10709668 DOI： 10.1007/0-306-46818-2_46
Source DB: PubMed Journal: Adv Exp Med Biol ISSN： 0065-2598 Impact factor: 2.622