Literature DB >> 10707143

Weill-Marchesani syndrome in three generations.

C Evereklioglu1, I F Hepsen, H Er.   

Abstract

BACKGROUND: Weill-Marchesani syndrome is a rare systemic connective tissue disorder consisting of brachymorphy, brachydactyly, ectopia lentis, spherophakia and glaucoma.
METHODS: We report 6 patients with Weill-Marchesani syndrome (with or without ocular involvement) in three generations, identified by screening 26 members of two families. This is the largest family in the literature showing an autosomal dominant pattern of inheritance.
RESULTS: Presenile vitreous liquefaction was present in all the younger cases. Weill-Marchesani syndrome was full-blown in two cases in the third generation, in which asymmetrical axial length and glaucomatous damage were present. To our knowledge this is the first report regarding asymmetrical axial length and glaucomatous damage, and presenile vitreous liquefaction in Weill-Marchesani syndrome with or without ocular involvement.
CONCLUSIONS: The longer axial length might be the precursor of impending severe glaucomatous damage. Presenile vitreous liquefaction in subtle young cases should alert the physician to the diagnosis of Weill-Marchesani syndrome on screening of the family members.

Entities:  

Mesh:

Year:  1999        PMID: 10707143     DOI: 10.1038/eye.1999.226

Source DB:  PubMed          Journal:  Eye (Lond)        ISSN: 0950-222X            Impact factor:   3.775


  6 in total

1.  Homozygous mutations in ADAMTS10 and ADAMTS17 cause lenticular myopia, ectopia lentis, glaucoma, spherophakia, and short stature.

Authors:  Jose Morales; Latifa Al-Sharif; Dania S Khalil; Jameela M A Shinwari; Prashant Bavi; Rahima A Al-Mahrouqi; Ali Al-Rajhi; Fowzan S Alkuraya; Brian F Meyer; Nada Al Tassan
Journal:  Am J Hum Genet       Date:  2009-11       Impact factor: 11.025

2.  Angle closure in younger patients.

Authors:  Brian M Chang; Jeffrey M Liebmann; Robert Ritch
Journal:  Trans Am Ophthalmol Soc       Date:  2002

3.  Missense mutations in FBN1 exons 41 and 42 cause Weill-Marchesani syndrome with thoracic aortic disease and Marfan syndrome.

Authors:  Alana Cecchi; Naomi Ogawa; Hugo R Martinez; Alicia Carlson; Yuxin Fan; Daniel J Penny; Dong-chuan Guo; Steven Eisenberg; Hazim Safi; Anthony Estrera; Richard A Lewis; Deborah Meyers; Dianna M Milewicz
Journal:  Am J Med Genet A       Date:  2013-07-29       Impact factor: 2.802

4.  Acromelic dysplasias: how rare musculoskeletal disorders reveal biological functions of extracellular matrix proteins.

Authors:  Sarah Stanley; Zerina Balic; Dirk Hubmacher
Journal:  Ann N Y Acad Sci       Date:  2020-09-02       Impact factor: 5.691

5.  Tumour necrosis factor alpha, lipid peroxidation and NO* are increased and associated with decreased free-radical scavenging enzymes in patients with Weill-Marchesani syndrome.

Authors:  Cem Evereklioglu; Yusuf Turkoz; Mustafa Calis; Fuat Duygulu; Aysun B Karabulut
Journal:  Mediators Inflamm       Date:  2004-06       Impact factor: 4.711

6.  Distribution of axial length in Chinese congenital ectopia lentis patients: a retrospective study.

Authors:  Yichi Zhang; Guangming Jin; Qianzhong Cao; Junxiong Lin; Jianqiang Lin; Yiyao Wang; Su Ern Poh; Charlotte Aimee Young; Danying Zheng
Journal:  BMC Ophthalmol       Date:  2017-07-03       Impact factor: 2.209

  6 in total

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