Literature DB >> 10702387

Hereditary hemorrhagic telangiectasia: A model for blood vessel growth and enlargement.

B S Jacobson1.   

Abstract

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Year:  2000        PMID： 10702387      PMCID： PMC1876865          DOI： 10.1016/S0002-9440(10)64939-5

Source DB:  PubMed          Journal:  Am J Pathol        ISSN： 0002-9440            Impact factor:   4.307


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  38 in total

Review 1.  Hereditary haemorrhagic telangiectasia and pulmonary arteriovenous malformations: issues in clinical management and review of pathogenic mechanisms.

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Review 5.  TGF-beta and the endothelium during immune injury.

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6.  Defective angiogenesis in mice lacking endoglin.

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7.  Mutant endoglin in hereditary hemorrhagic telangiectasia type 1 is transiently expressed intracellularly and is not a dominant negative.

Authors:  N Pece; S Vera; U Cymerman; R I White; J L Wrana; M Letarte
Journal:  J Clin Invest       Date:  1997-11-15       Impact factor: 14.808

8.  Endoglin is an accessory protein that interacts with the signaling receptor complex of multiple members of the transforming growth factor-beta superfamily.

Authors:  N P Barbara; J L Wrana; M Letarte
Journal:  J Biol Chem       Date:  1999-01-08       Impact factor: 5.157

9.  Mapping of vascular dendritic cells in atherosclerotic arteries suggests their involvement in local immune-inflammatory reactions.

Authors:  Y V Bobryshev; R S Lord
Journal:  Cardiovasc Res       Date:  1998-03       Impact factor: 10.787

Review 10.  TGF-beta signal transduction.

Authors:  J Massagué
Journal:  Annu Rev Biochem       Date:  1998       Impact factor: 23.643
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  3 in total

Review 1.  Angiogenesis-dependent diseases and angiogenesis therapy.

Authors:  J Tímár; B Döme; K Fazekas; A Janovics ; S Paku
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2.  Spontaneous regression of cerebral arteriovenous malformation in hereditary hemorrhagic telangiectasia.

Authors:  Harry J Cloft
Journal:  AJNR Am J Neuroradiol       Date:  2002 Jun-Jul       Impact factor: 3.825

Review 3.  Endoglin: An 'Accessory' Receptor Regulating Blood Cell Development and Inflammation.

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