Literature DB >> 10652465

Rheumatoid arthritis and p53: how oxidative stress might alter the course of inflammatory diseases.

P P Tak1, N J Zvaifler, D R Green, G S Firestein.   

Abstract

Oxidative stress at sites of chronic inflammation can cause permanent genetic changes. The development of mutations in the p53 tumor suppressor gene and other key regulatory genes could help convert inflammation into chronic disease in rheumatoid arthritis and other inflammatory disorders.

Entities:  

Mesh：

Substances：

Year:  2000        PMID： 10652465     DOI： 10.1016/s0167-5699(99)01552-2

Source DB:  PubMed          Journal:  Immunol Today        ISSN： 0167-5699


  86 in total

1.  Analysis of synovial biopsy samples: opportunities and challenges.

Authors:  P P Tak
Journal:  Ann Rheum Dis       Date:  2000-12       Impact factor: 19.103

2.  Identification of the advanced glycation end products N(epsilon)-carboxymethyllysine in the synovial tissue of patients with rheumatoid arthritis.

Authors:  S Drinda; S Franke; C C Canet; P Petrow; R Bräuer; C Hüttich; G Stein; G Hein
Journal:  Ann Rheum Dis       Date:  2002-06       Impact factor: 19.103

3.  Functional analysis of the p53 codon 72 polymorphism in black South Africans with rheumatoid arthritis--a pilot study.

Authors:  Devapregasan Moodley; Girish M Mody; Anil A Chuturgoon
Journal:  Clin Rheumatol       Date:  2010-06-10       Impact factor: 2.980

4.  Analysis of the cell infiltrate and expression of matrix metalloproteinases and granzyme B in paired synovial biopsy specimens from the cartilage-pannus junction in patients with RA.

Authors:  T J Smeets; M C Kraan; S Galjaard; P P Youssef; M D Smith; P P Tak
Journal:  Ann Rheum Dis       Date:  2001-06       Impact factor: 19.103

5.  The p53 status in juvenile chronic arthritis and rheumatoid arthritis.

Authors:  H Taubert; B Thamm; A Meye; F Bartel; A K Rost; D Heidenreich; V John; J Brandt; M Bache; P Würl; H Schmidt; D Riemann
Journal:  Clin Exp Immunol       Date:  2000-11       Impact factor: 4.330

6.  Association between activity and genotypes of paraoxonase1 L55M (rs854560) increases the disease activity of rheumatoid arthritis through oxidative stress.

Authors:  Maryam Tanhapour; Shiva Shahmohamadnejad; Asad Vaisi-Raygani; Amir Kiani; Yadola Shakiba; Zohreh Rahimi; Fariborz Bahrehmand; Ebrahim Shakiba; Ali-Akbar Vaisi-Raygani; Reza Alibakhshi; Atefeh Eivazi; Tayebeh Pourmotabbed
Journal:  Mol Biol Rep       Date:  2018-12-01       Impact factor: 2.316

7.  Silencing the expression of Ras family GTPase homologues decreases inflammation and joint destruction in experimental arthritis.

Authors:  Daphne de Launay; Jeroen Vreijling; Linda M Hartkamp; Olga N Karpus; Joana R F Abreu; Marjolein A van Maanen; Marjolein E Sanders; Aleksander M Grabiec; Jörg Hamann; Henrik Ørum; Margriet J Vervoordeldonk; Kees Fluiter; Paul P Tak; Kris A Reedquist
Journal:  Am J Pathol       Date:  2010-10-22       Impact factor: 4.307

Review 8.  Stress, oxidative injury and disease.

Authors:  Kaushal K Srivastava; Ratan Kumar
Journal:  Indian J Clin Biochem       Date:  2014-06-01

9.  Fanconi anemia proteins and endogenous stresses.

Authors:  Qishen Pang; Paul R Andreassen
Journal:  Mutat Res       Date:  2009-07-31       Impact factor: 2.433

10.  The effect of anti-TNF agent on oxidation status in patients with ankylosing spondylitis.

Authors:  Murat Karkucak; Erhan Capkin; Ahmet Alver; Ayse Akyuz; Abdulkadir Kiris; Emel Ak; Murat Topbas; Mehmet Tosun
Journal:  Clin Rheumatol       Date:  2009-12-11       Impact factor: 2.980
View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.