Literature DB >> 10606361

Inpatient treatment of rheumatoid arthritis with synacthen depot: a double blind placebo controlled trial with 6 month followup.

W J Taylor1, C N Rajapakse, K A Harris, A A Harrison, M M Corkill.   

Abstract

OBJECTIVE: To assess the additional benefit of synacthen depot over standard inpatient care for patients hospitalized with active rheumatoid arthritis (RA).
METHODS: All patients admitted to our unit with active RA without exclusion criteria were invited to participate and randomized to subcutaneous synacthen depot 0.5 mg on alternate days for 2 injections or 2 injections of saline. Patients, staff, and assessors of response were blinded to the intervention. Assessment [OMERACT set, American College of Rheumatology (ACR) global improvement, dose of intraarticular (IA) or intramuscular (IM) methylprednisolone] was performed at admission to hospital, at discharge, and at 3 and 6 months. Oral prednisone use constituted a protocol violation.
RESULTS: Of 137 patients with RA admitted over the period of recruitment, 36 (26%) were enrolled; 31 completed followup. There were no between-group differences in the change from admission of any individual disease activity measure at any time point. However, using a rigorous global response measure (ACR 50%), a difference was detected in favor of synacthen depot at discharge (52.6% of the intervention group improved vs. 17.6% of controls; p = 0.029, number-needed-to-treat 2.86). Patients treated with synacthen depot showed a trend toward more IA or IM corticosteroid between discharge and 3 months (mean dose 56 vs. 31 mg; p = 0.19) and a trend toward more patients requiring a change in slow acting antirheumatic drug after discharge (4 vs. 1; p = 0.27).
CONCLUSION: There is some additional benefit of synacthen depot in the hospital treatment of RA, but the effect is lost by 3 months, with a suggestion of rebound worsening in these patients. We postulate that oversuppression of corticotrophin releasing hormone by exogenous adrenocorticotrophic hormone in patients who already have a hypothalamic deficit may contribute to the rebound worsening of disease activity seen in these patients.

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Year:  1999        PMID: 10606361

Source DB:  PubMed          Journal:  J Rheumatol        ISSN: 0315-162X            Impact factor:   4.666


  4 in total

Review 1.  Current view of glucocorticoid co-therapy with DMARDs in rheumatoid arthritis.

Authors:  Jos N Hoes; Johannes W G Jacobs; Frank Buttgereit; Johannes W J Bijlsma
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2.  Curbing Inflammation through Endogenous Pathways: Focus on Melanocortin Peptides.

Authors:  Tazeen J Ahmed; Trinidad Montero-Melendez; Mauro Perretti; Costantino Pitzalis
Journal:  Int J Inflam       Date:  2013-05-07

3.  Cortisol Response in Healthy and Diseased Dogs after Stimulation with a Depot Formulation of Synthetic ACTH.

Authors:  N S Sieber-Ruckstuhl; W A Burkhardt; N Hofer-Inteeworn; B Riond; I T Rast; R Hofmann-Lehmann; C E Reusch; F S Boretti
Journal:  J Vet Intern Med       Date:  2015-10-27       Impact factor: 3.333

4.  The efficacy of systemic glucocorticosteroids for pain in rheumatoid arthritis: a systematic literature review and meta-analysis.

Authors:  Daniel F McWilliams; Divya Thankaraj; Julie Jones-Diette; Rheinallt Morgan; Onosi S Ifesemen; Nicholas G Shenker; David A Walsh
Journal:  Rheumatology (Oxford)       Date:  2021-12-24       Impact factor: 7.580

  4 in total

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