Literature DB >> 10587654

SMAD destruction turns off signalling.

C H Heldin, P ten Dijke.   

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Year:  1999        PMID： 10587654     DOI： 10.1038/70223

Source DB:  PubMed          Journal:  Nat Cell Biol        ISSN： 1465-7392            Impact factor:   28.824


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  4 in total

1.  Transforming growth factor beta-independent shuttling of Smad4 between the cytoplasm and nucleus.

Authors:  C E Pierreux; F J Nicolás; C S Hill
Journal:  Mol Cell Biol       Date:  2000-12       Impact factor: 4.272

2.  Insulin-like growth factor-binding protein-3 promotes transforming growth factor-{beta}1-mediated epithelial-to-mesenchymal transition and motility in transformed human esophageal cells.

Authors:  Mitsuteru Natsuizaka; Shinya Ohashi; Gabrielle S Wong; Azal Ahmadi; Ross A Kalman; Daniela Budo; Andres J Klein-Szanto; Meenhard Herlyn; J Alan Diehl; Hiroshi Nakagawa
Journal:  Carcinogenesis       Date:  2010-05-31       Impact factor: 4.944

3.  Ligand-dependent ubiquitination of Smad3 is regulated by casein kinase 1 gamma 2, an inhibitor of TGF-beta signaling.

Authors:  X Guo; D S Waddell; W Wang; Z Wang; N T Liberati; S Yong; X Liu; X-F Wang
Journal:  Oncogene       Date:  2008-09-15       Impact factor: 9.867

4.  Axin and GSK3- control Smad3 protein stability and modulate TGF- signaling.

Authors:  Xing Guo; Alejandro Ramirez; David S Waddell; Zhizhong Li; Xuedong Liu; Xiao-Fan Wang
Journal:  Genes Dev       Date:  2008-01-01       Impact factor: 11.361
  4 in total

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