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27 in total

1. Mutational analysis of the candidate tumor suppressor genes TEL and KIP1 in childhood acute lymphoblastic leukemia.

Authors: K Stegmaier; S Takeuchi; T R Golub; S K Bohlander; C R Bartram; H P Koeffler
Journal: Cancer Res Date: 1996-03-15 Impact factor: 12.701

2. Increased proteasome-dependent degradation of the cyclin-dependent kinase inhibitor p27 in aggressive colorectal carcinomas.

Authors: M Loda; B Cukor; S W Tam; P Lavin; M Fiorentino; G F Draetta; J M Jessup; M Pagano
Journal: Nat Med Date: 1997-02 Impact factor: 53.440

3. Kip/Cip and Ink4 Cdk inhibitors cooperate to induce cell cycle arrest in response to TGF-beta.

Authors: I Reynisdóttir; K Polyak; A Iavarone; J Massagué
Journal: Genes Dev Date: 1995-08-01 Impact factor: 11.361

4. Mice lacking p27(Kip1) display increased body size, multiple organ hyperplasia, retinal dysplasia, and pituitary tumors.

Authors: K Nakayama; N Ishida; M Shirane; A Inomata; T Inoue; N Shishido; I Horii; D Y Loh; K Nakayama
Journal: Cell Date: 1996-05-31 Impact factor: 41.582

The p27/Kip1 locus shows no loss of heterozygosity in human pituitary adenomas.

1. Mutational analysis of the candidate tumor suppressor genes TEL and KIP1 in childhood acute lymphoblastic leukemia.

2. Increased proteasome-dependent degradation of the cyclin-dependent kinase inhibitor p27 in aggressive colorectal carcinomas.

3. Kip/Cip and Ink4 Cdk inhibitors cooperate to induce cell cycle arrest in response to TGF-beta.

4. Mice lacking p27(Kip1) display increased body size, multiple organ hyperplasia, retinal dysplasia, and pituitary tumors.

5. Enhanced growth of mice lacking the cyclin-dependent kinase inhibitor function of p27(Kip1).

6. Mutational analysis of the human cyclin-dependent kinase inhibitor p27kip1 in primary breast carcinomas.

7. Mutation and cancer: statistical study of retinoblastoma.

8. Cyclic AMP-induced G1 phase arrest mediated by an inhibitor (p27Kip1) of cyclin-dependent kinase 4 activation.

9. p27Kip1: chromosomal mapping to 12p12-12p13.1 and absence of mutations in human tumors.

10. Role of the ubiquitin-proteasome pathway in regulating abundance of the cyclin-dependent kinase inhibitor p27.

1. Modeling sporadic loss of heterozygosity in mice by using mosaic analysis with double markers (MADM).