Literature DB >> 1057776

Gastric pathology produced by hypothalamic lesions in rats.

E Lindholm, G S Shumway, C V Grijalva, T Schallert, M Ruppel.   

Abstract

Rats made aphagic and adipsic by damage to the lateral hypothalamus develop gastric lesions and lose body weight excessively over a four day period. Control observations indicate that these effects cannot be accounted for by food and water deprivation or by operative trauma. The possibility that gastric lesions may contribute to aphagia or anorexia is discussed.

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Year:  1975        PMID: 1057776     DOI: 10.1016/0031-9384(75)90161-4

Source DB:  PubMed          Journal:  Physiol Behav        ISSN: 0031-9384


  3 in total

Review 1.  Neurobiologic and psychobiologic mechanisms in gastric function and ulceration.

Authors:  H Weiner; D Novin; C V Grijalva; Y Taché; T Garrick
Journal:  West J Med       Date:  1985-08

2.  Excitotoxic lesions of the lateral hypothalamus made by N-methyl-d-aspartate in the rat: behavioural, histological and biochemical analyses.

Authors:  P Winn; A Clark; M Hastings; J Clark; M Latimer; E Rugg; B Brownlee
Journal:  Exp Brain Res       Date:  1990       Impact factor: 1.972

3.  Effects of gastric vagotomy on visceral cell proliferation induced by ventromedial hypothalamic lesions: role of vagal hyperactivity.

Authors:  Yuri Kintaka; Toshimasa Osaka; Yoko Suzuki; Takeo Hashiguchi; Akira Niijima; Haruaki Kageyama; Takenoya Fumiko; Seiji Shioda; Shuji Inoue
Journal:  J Mol Neurosci       Date:  2009-05-20       Impact factor: 3.444

  3 in total

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