Literature DB >> 10577561

Adenosine as an adjunct to thrombolytic therapy for acute myocardial infarction: results of a multicenter, randomized, placebo-controlled trial: the Acute Myocardial Infarction STudy of ADenosine (AMISTAD) trial.

K W Mahaffey1, J A Puma, N A Barbagelata, M F DiCarli, M A Leesar, K F Browne, P R Eisenberg, R Bolli, A C Casas, V Molina-Viamonte, C Orlandi, R Blevins, R J Gibbons, R M Califf, C B Granger.   

Abstract

OBJECTIVES: The Acute Myocardial Infarction STudy of ADenosine (AMISTAD) trial was designed to test the hypothesis that adenosine as an adjunct to thrombolysis would reduce myocardial infarct size.
BACKGROUND: Reperfusion therapy for acute myocardial infarction (MI) has been shown to reduce mortality, but reperfusion itself also may have deleterious effects.
METHODS: The AMISTAD trial was a prospective, open-label trial of thrombolysis with randomization to adenosine or placebo in 236 patients within 6 h of infarction onset. The primary end point was infarct size as determined by Tc-99 m sestamibi single-photon emission computed tomography (SPECT) imaging 6+/-1 days after enrollment based on multivariable regression modeling to adjust for covariates. Secondary end points were myocardial salvage index and a composite of in-hospital clinical outcomes (death, reinfarction, shock, congestive heart failure or stroke).
RESULTS: In all, 236 patients were enrolled. Final infarct size was assessed in 197 (83%) patients. There was a 33% relative reduction in infarct size (p = 0.03) with adenosine. There was a 67% relative reduction in infarct size in patients with anterior infarction (15% in the adenosine group vs. 45.5% in the placebo group) but no reduction in patients with infarcts located elsewhere (11.5% for both groups). Patients randomized to adenosine tended to reach the composite clinical end point more often than those assigned to placebo (22% vs. 16%; odds ratio, 1.43; 95% confidence interval, 0.71 to 2.89).
CONCLUSIONS: Many agents thought to attenuate reperfusion injury have been unsuccessful in clinical investigation. In this study, adenosine resulted in a significant reduction in infarct size. These data support the need for a large clinical outcome trial.

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Year:  1999        PMID: 10577561     DOI: 10.1016/s0735-1097(99)00418-0

Source DB:  PubMed          Journal:  J Am Coll Cardiol        ISSN: 0735-1097            Impact factor:   24.094


  94 in total

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Authors:  D M Yellon; G F Baxter
Journal:  Heart       Date:  2000-04       Impact factor: 5.994

Review 2.  Therapeutic potential of ischaemic preconditioning.

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Review 3.  CMR for characterization of the myocardium in acute coronary syndromes.

Authors:  Erica Dall'Armellina; Theodoros D Karamitsos; Stefan Neubauer; Robin P Choudhury
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4.  Playing hide and seek with adenosine receptors.

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5.  Adenosine receptor crossroads in sickle cell disease.

Authors:  Mark T Gladwin
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Review 6.  Pathogenesis of myocardial ischemia-reperfusion injury and rationale for therapy.

Authors:  Aslan T Turer; Joseph A Hill
Journal:  Am J Cardiol       Date:  2010-08-01       Impact factor: 2.778

7.  Tc-99m sestamibi infarct size as a surrogate endpoint.

Authors:  Raymond J Gibbons; Todd D Miller
Journal:  J Nucl Cardiol       Date:  2005 Jan-Feb       Impact factor: 5.952

8.  Overexpression of A(3) adenosine receptors decreases heart rate, preserves energetics, and protects ischemic hearts.

Authors:  Heather R Cross; Elizabeth Murphy; Richard G Black; John Auchampach; Charles Steenbergen
Journal:  Am J Physiol Heart Circ Physiol       Date:  2002-06-20       Impact factor: 4.733

Review 9.  Therapeutic receptor targets of ischemic preconditioning.

Authors:  Ryan M Fryer; John A Auchampach; Garrett J Gross
Journal:  Cardiovasc Res       Date:  2002-08-15       Impact factor: 10.787

10.  Adenosine myocardial perfusion single photon emission computed tomographic stress testing 24-72 h after uncomplicated myocardial infarction.

Authors:  Jan Kulhanek; Vincent L Sorrell; Reza E Ershadi; Brian R Cabarrus; Douglas B Short; Assad Movahed
Journal:  Int J Cardiovasc Imaging       Date:  2002-08       Impact factor: 2.357

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