Literature DB >> 10553496

Dinucleotide repeat polymorphisms within the Flt-1 gene in minimal change nephropathy.

R G Parry1, K M Gillespie, A G Clark, P W Mathieson.   

Abstract

Dysregulation of Flt-1, a major receptor for vascular permeability factor (VPF), may provide a mechanism for the development of proteinuria in minimal change nephropathy (MCN). The gene for Flt-1 has a polymorphic dinucleotide repeat. We have demonstrated an 88% predominance of one allele and an 80% rate of homozygosity for this polymorphism with no association with MCN.

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Year:  1999        PMID: 10553496     DOI: 10.1046/j.1365-2370.1999.00161.x

Source DB:  PubMed          Journal:  Eur J Immunogenet        ISSN: 0960-7420


  3 in total

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Authors:  Atis Muehlenbachs; Michal Fried; Jeff Lachowitzer; Theonest K Mutabingwa; Patrick E Duffy
Journal:  Proc Natl Acad Sci U S A       Date:  2008-09-08       Impact factor: 11.205

2.  The Effect of FLT1 Variant on Long-Term Cardiovascular Outcomes: Validation of a Locus Identified in a Previous Genome-Wide Association Study.

Authors:  Chan Joo Lee; Ji-Young Lee; Chi-Yoon Oum; Jong-Chan Youn; Seok-Min Kang; Donghoon Choi; Yangsoo Jang; Sungha Park; Sun Ha Jee; Sang-Hak Lee
Journal:  PLoS One       Date:  2016-10-13       Impact factor: 3.240

3.  Dinucleotide repeat polymorphism in Fms-like tyrosine kinase-1 (Flt-1) gene is not associated with preeclampsia.

Authors:  Shin-Young Kim; Ji-Hyae Lim; Jae-Hyug Yang; Moon-Young Kim; Jung-Yeol Han; Hyun-Kyong Ahn; Jun-Seek Choi; So-Yeon Park; Mi-Jin Kim; Hyun-Mee Ryu
Journal:  BMC Med Genet       Date:  2008-07-17       Impact factor: 2.103

  3 in total

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