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Literature DB >> 10521080

Extracellular proteolysis: new paradigms for an old paradox.

C A Owen¹, E J Campbell.

Abstract

Mesh：

Substances：
Endopeptidases

Year: 1999 PMID： 10521080 DOI： 10.1016/s0022-2143(99)90148-8

Source DB: PubMed Journal: J Lab Clin Med ISSN： 0022-2143

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Cited

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9 in total

Review 1. Proteinases and oxidants as targets in the treatment of chronic obstructive pulmonary disease.

Authors: Caroline A Owen
Journal: Proc Am Thorac Soc Date: 2005

2. Matrix metalloproteinase-9 deficiency protects mice from severe influenza A viral infection.

Authors: Joselyn Rojas-Quintero; Xiaoyun Wang; Jennifer Tipper; Patrick R Burkett; Joaquin Zuñiga; Amit R Ashtekar; Francesca Polverino; Amit Rout; Ilyas Yambayev; Carmen Hernández; Luis Jimenez; Gustavo Ramírez; Kevin S Harrod; Caroline A Owen
Journal: JCI Insight Date: 2018-12-20

3. Neutrophil serine proteases exert proteolytic activity on endothelial cells.

Authors: Uwe Jerke; Daniel Perez Hernandez; Patrick Beaudette; Brice Korkmaz; Gunnar Dittmar; Ralph Kettritz
Journal: Kidney Int Date: 2015-06-10 Impact factor: 10.612

4. ADAM9 is a novel product of polymorphonuclear neutrophils: regulation of expression and contributions to extracellular matrix protein degradation during acute lung injury.

Authors: Robin Roychaudhuri; Anja H Hergrueter; Francesca Polverino; Maria E Laucho-Contreras; Kushagra Gupta; Niels Borregaard; Caroline A Owen
Journal: J Immunol Date: 2014-07-25 Impact factor: 5.422

Extracellular proteolysis: new paradigms for an old paradox.

Review 1. Proteinases and oxidants as targets in the treatment of chronic obstructive pulmonary disease.

2. Matrix metalloproteinase-9 deficiency protects mice from severe influenza A viral infection.

3. Neutrophil serine proteases exert proteolytic activity on endothelial cells.

4. ADAM9 is a novel product of polymorphonuclear neutrophils: regulation of expression and contributions to extracellular matrix protein degradation during acute lung injury.

Review 5. Cathepsin G: roles in antigen presentation and beyond.

6. A Proteinase 3 Contribution to Juvenile Idiopathic Arthritis-Associated Cartilage Damage.

7. Systemic inflammation in COPD in relation to smoking status.

8. Inflammatory markers in chronic obstructive pulmonary disease patients with different α1 antitrypsin genotypes.

9. Proteinase 3 contributes to endothelial dysfunction in an experimental model of sepsis.