Literature DB >> 10510162

Forearm reactive hyperaemia is not mediated by nitric oxide in healthy volunteers.

A G Nugent1, C McGurk, D McAuley, S Maguire, B Silke, G D Johnston.   

Abstract

AIMS: To determine the role of nitric oxide (NO) in forearm reactive hyperaemia in healthy human subjects.
METHODS: Ten healthy subjects aged 19-34 years underwent brachial artery cannulation. Forearm circulatory arrest was achieved by means of an upper arm cuff inflated to 200 mmHg for 5 min. The blood flow responses during reactive hyperaemia were measured using venous occlusion plethysmography following a 10 min intra-arterial infusion of 8 micromol min-1 N-monomethyl L-arginine (L-NMMA) and following matching placebo administered in random order. Results were analysed by repeated measures anova and t-tests.
RESULTS: L-NMMA resulted in a significant reduction of basal forearm blood flow indicating inhibition of basal NO release (P=0.005). There was no significant difference between the blood flow responses during reactive hyperaemia following L-NMMA and placebo (P=0.97).
CONCLUSIONS: Nitric oxide production does not make a significant contribution to the vasodilatation associated with reactive hyperaemia in the human forearm.

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Year:  1999        PMID: 10510162      PMCID: PMC2014319          DOI: 10.1046/j.1365-2125.1999.00032.x

Source DB:  PubMed          Journal:  Br J Clin Pharmacol        ISSN: 0306-5251            Impact factor:   4.335


  16 in total

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  2 in total

1.  Positional differences in reactive hyperemia provide insight into initial phase of exercise hyperemia.

Authors:  Jeffrey L Jasperse; J Kevin Shoemaker; Eric J Gray; Philip S Clifford
Journal:  J Appl Physiol (1985)       Date:  2015-07-02

2.  Reactive hyperemia occurs via activation of inwardly rectifying potassium channels and Na+/K+-ATPase in humans.

Authors:  Anne R Crecelius; Jennifer C Richards; Gary J Luckasen; Dennis G Larson; Frank A Dinenno
Journal:  Circ Res       Date:  2013-08-12       Impact factor: 17.367

  2 in total

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