| Literature DB >> 10491738 |
Abstract
The physiological role of 24,25-dihydroxyvitamin D remains controversial. Recent results suggest that 24,25-dihydroxyvitamin D is essential for fracture healing, and binding sites for 24,25-dihydroxyvitamin D have been identified in fracture callus tissue. Mice deficient in the 25-hydroxyvitamin D-24-hydroxylase enzyme provide novel genetic tools in which to study the role of 24,25-dihydroxyvitamin D in bone development and fracture repair.Entities:
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Year: 1999 PMID： 10491738 DOI： 10.1097/00041552-199907000-00007
Source DB: PubMed Journal: Curr Opin Nephrol Hypertens ISSN： 1062-4821 Impact factor: 2.894