Literature DB >> 10468412

T cells: pathogenic cells and therapeutic targets in rheumatoid arthritis.

D E Yocum1.   

Abstract

OBJECTIVES: To provide: 1) a brief review of current thought on the role of T cells in the pathogenesis of rheumatoid arthritis (RA); and 2) To provide an overview of RA therapies directed against T cells.
METHODS: The following papers in relevant American and European medical journals were reviewed. Those related to: the role of T cells in the pathogenesis of RA; to biological therapy directed against cell surface markers specific to T cell populations implicated in RA; and to treatment of RA with cyclosporin A and leflunomide, pharmacological agents known to interfere with the T cell response to antigens.
RESULTS: Although a variety of cell types are now recognized as contributors to the progressive joint destruction that is a hallmark of RA, T cell activation is still thought to be a central event in the initiation and progression of this disease. As a result, various therapeutic options directed against T cells have been developed. These include biological agents directed against specific populations of activated T cells and pharmacological agents that have specific T cell-modulatory actions.
CONCLUSIONS: Use of T cell-directed biological therapies for RA has been disappointing, as a result of both lack of efficacy and serious toxicity. Treatment of RA with pharmacological agents that interfere with antigen-driven T cell proliferation has been more successful.

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Year:  1999        PMID: 10468412     DOI: 10.1016/s0049-0172(99)80035-3

Source DB:  PubMed          Journal:  Semin Arthritis Rheum        ISSN: 0049-0172            Impact factor:   5.532


  8 in total

1.  Inducible nitric oxide synthase is expressed in synovial fluid granulocytes.

Authors:  J Cedergren; T Forslund; T Sundqvist; T Skogh
Journal:  Clin Exp Immunol       Date:  2002-10       Impact factor: 4.330

2.  Neuronal survival after CNS insult is determined by a genetically encoded autoimmune response.

Authors:  J Kipnis; E Yoles; H Schori; E Hauben; I Shaked; M Schwartz
Journal:  J Neurosci       Date:  2001-07-01       Impact factor: 6.167

3.  Interleukin 17 synergises with tumour necrosis factor alpha to induce cartilage destruction in vitro.

Authors:  R L Van Bezooijen; L Van Der Wee-Pals; S E Papapoulos; C W G M Löwik
Journal:  Ann Rheum Dis       Date:  2002-10       Impact factor: 19.103

Review 4.  The role of the T cell in autoimmune inflammation.

Authors:  Alla Skapenko; Jan Leipe; Peter E Lipsky; Hendrik Schulze-Koops
Journal:  Arthritis Res Ther       Date:  2005-03-16       Impact factor: 5.156

5.  Increased activation-induced cell death in peripheral lymphocytes of rheumatoid arthritis patients: the mechanism of action.

Authors:  Xiaolei Tang; David E Yocum; David Dejonghe; Kathryn Nordensson; Douglas F Lake; John Richard
Journal:  Immunology       Date:  2004-07       Impact factor: 7.397

6.  Suppressive effect of combination treatment of leflunomide and methotrexate on chemokine expression in patients with rheumatoid arthritis.

Authors:  C Y Ho; C K Wong; E K Li; L S Tam; C W K Lam
Journal:  Clin Exp Immunol       Date:  2003-07       Impact factor: 4.330

7.  Joint erosion in rheumatoid arthritis: interactions between tumour necrosis factor alpha, interleukin 1, and receptor activator of nuclear factor kappaB ligand (RANKL) regulate osteoclasts.

Authors:  D O' Gradaigh; D Ireland; S Bord; J E Compston
Journal:  Ann Rheum Dis       Date:  2004-04       Impact factor: 19.103

8.  FGF2 cooperates with IL-17 to promote autoimmune inflammation.

Authors:  Xinrui Shao; Siyuan Chen; Daping Yang; Mengtao Cao; Yikun Yao; Zhengxi Wu; Ningli Li; Nan Shen; Xiaoxia Li; Xinyang Song; Youcun Qian
Journal:  Sci Rep       Date:  2017-08-01       Impact factor: 4.379

  8 in total

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