Literature DB >> 10407118

Persistent activation of calcium-activated and calcium-independent protein kinase C in response to electrical afterdischarge from peptidergic neurons of aplysia.

N L Wayne1, W Lee, Y J Kim.   

Abstract

The purpose of this work was to investigate the effects of electrical afterdischarge on protein kinase C (PKC) activity from bag cell neurons (BCNs) of Aplysia. Bilateral clusters of BCNs were divided: one cluster was stimulated to afterdischarge, the other was a control. Clusters were processed for PKC activity assay 5-120 min after electrical stimulation. Afterdischarge triggered a rapid and persistent increase in both calcium-activated and calcium-independent PKC activity. Copyright 1999 Elsevier Science B. V.

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Year:  1999        PMID： 10407118     DOI： 10.1016/s0006-8993(99)01594-2

Source DB:  PubMed          Journal:  Brain Res        ISSN： 0006-8993            Impact factor:   3.252


  9 in total

1.  Neurohormone secretion persists after post-afterdischarge membrane depolarization and cytosolic calcium elevation in peptidergic neurons in intact nervous tissue.

Authors:  Stephan Michel; Nancy L Wayne
Journal:  J Neurosci       Date:  2002-10-15       Impact factor: 6.167

2.  Regulation of an Aplysia bag-cell neuron cation channel by closely associated protein kinase A and a protein phosphatase.

Authors:  Neil S Magoski
Journal:  J Neurosci       Date:  2004-07-28       Impact factor: 6.167

3.  Association/dissociation of a channel-kinase complex underlies state-dependent modulation.

Authors:  Neil S Magoski; Leonard K Kaczmarek
Journal:  J Neurosci       Date:  2005-08-31       Impact factor: 6.167

4.  PKC-induced intracellular trafficking of Ca(V)2 precedes its rapid recruitment to the plasma membrane.

Authors:  Yalan Zhang; Jessica S Helm; Adriano Senatore; J David Spafford; Leonard K Kaczmarek; Elizabeth A Jonas
Journal:  J Neurosci       Date:  2008-03-05       Impact factor: 6.167

5.  Protein kinase modulation of a neuronal cation channel requires protein-protein interactions mediated by an Src homology 3 domain.

Authors:  Neil S Magoski; Gisela F Wilson; Leonard K Kaczmarek
Journal:  J Neurosci       Date:  2002-01-01       Impact factor: 6.167

6.  PKC enhances the capacity for secretion by rapidly recruiting covert voltage-gated Ca2+ channels to the membrane.

Authors:  Christopher J Groten; Neil S Magoski
Journal:  J Neurosci       Date:  2015-02-11       Impact factor: 6.167

7.  Ca2+ removal by the plasma membrane Ca2+-ATPase influences the contribution of mitochondria to activity-dependent Ca2+ dynamics in Aplysia neuroendocrine cells.

Authors:  Christopher J Groten; Jonathan T Rebane; Heather M Hodgson; Alamjeet K Chauhan; Gunnar Blohm; Neil S Magoski
Journal:  J Neurophysiol       Date:  2016-02-10       Impact factor: 2.714

8.  A Closely Associated Phospholipase C Regulates Cation Channel Function through Phosphoinositide Hydrolysis.

Authors:  Raymond M Sturgeon; Neil S Magoski
Journal:  J Neurosci       Date:  2018-07-23       Impact factor: 6.167

9.  Hydrogen Peroxide Gates a Voltage-Dependent Cation Current in Aplysia Neuroendocrine Cells.

Authors:  Alamjeet K Chauhan; Neil S Magoski
Journal:  J Neurosci       Date:  2019-11-01       Impact factor: 6.167
  9 in total

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