Literature DB >> 10393064

Severe ocular abnormalities in C57BL/6 but not in 129/Sv p53-deficient mice.

S Ikeda1, N L Hawes, B Chang, C S Avery, R S Smith, P M Nishina.   

Abstract

PURPOSE: To demonstrate the importance of genetic background interaction on the development of ocular phenotypes in p53-deficient mice.
METHODS: Eyes of adult mice, homozygous and heterozygous for the p53 gene disruption in the 129/SvJ and C57BL/6J (B6) genetic backgrounds, and their F1 progeny were examined by indirect ophthalmoscopy and by light microscopy.
RESULTS: Indirect ophthalmoscopy revealed unilateral or bilateral vitreal opacities, fibrous retrolental tissue, and retinal folds in adult B6 mice but not in 129/Sv mice homozygous for a p53 null mutation. In B6 p53-/- mice, blood vessels extended from the peripapillary inner retina through the posterior vitreous and into the retrolental membrane. Optic nerves were hypoplastic.
CONCLUSIONS: These findings indicate that alleles from the B6 background contribute to the aberrant ocular phenotypes observed in p53 deficiency. They also suggest that p53 or the pathway in which it functions may be important for normal eye development.

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Year:  1999        PMID: 10393064

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  25 in total

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8.  Pathogenesis of persistent hyperplastic primary vitreous in mice lacking the arf tumor suppressor gene.

Authors:  Amy C Martin; J Derek Thornton; Jiewiu Liu; XiaoFei Wang; Jian Zuo; Monica M Jablonski; Edward Chaum; Frederique Zindy; Stephen X Skapek
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9.  p19(Arf) limits primary vitreous cell proliferation driven by PDGF-B.

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10.  N-methyl-N-nitrosourea-induced retinal degeneration in mice is independent of the p53 gene.

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