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Literature DB >> 10233780

UV fingerprint CDKN2a but no p14ARF mutations in sporadic melanomas.

K Peris, S Chimenti, M C Fargnoli, P Valeri, H Kerl, P Wolf.

Abstract

Entities: Disease Gene

Mesh：

Substances：

Year: 1999 PMID： 10233780 DOI： 10.1046/j.1523-1747.1999.00575.x

Source DB: PubMed Journal: J Invest Dermatol ISSN： 0022-202X Impact factor: 8.551

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Cited

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3 in total

1. Components of the Rb pathway are critical targets of UV mutagenesis in a murine melanoma model.

Authors: Karuppiah Kannan; Norman E Sharpless; Jin Xu; Ronan C O'Hagan; Marcus Bosenberg; Lynda Chin
Journal: Proc Natl Acad Sci U S A Date: 2003-01-21 Impact factor: 11.205

2. Towards a Better Understanding of the Molecular Mechanisms Involved in Sunlight-Induced Melanoma.

Authors: Mandy Williams; Allal Ouhtit
Journal: J Biomed Biotechnol Date: 2005

3. Loss of p16^INK4A stimulates aberrant mitochondrial biogenesis through a CDK4/Rb-independent pathway.

Authors: Ethika Tyagi; Bin Liu; Chelsea Li; Tong Liu; Jared Rutter; Douglas Grossman
Journal: Oncotarget Date: 2017-08-03

3 in total

UV fingerprint CDKN2a but no p14ARF mutations in sporadic melanomas.

1. Components of the Rb pathway are critical targets of UV mutagenesis in a murine melanoma model.

2. Towards a Better Understanding of the Molecular Mechanisms Involved in Sunlight-Induced Melanoma.

3. Loss of p16INK4A stimulates aberrant mitochondrial biogenesis through a CDK4/Rb-independent pathway.

3. Loss of p16^INK4A stimulates aberrant mitochondrial biogenesis through a CDK4/Rb-independent pathway.